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| Cas No.: | 4474-91-3 |
| Synonyms: | Angiotensin II;Hypertensin II;Ang II;DRVYIHPF |
| SMILES: | CC[C@H](C)[C@@H](C(=O)N[C@@H](CC1=CN=CN1)C(=O)N2CCC[C@H]2C(=O)N[C@@H](CC3=CC=CC=C3)C(=O)O)NC(=O)[C@H](CC4=CC=C(C=C4)O)NC(=O)[C@H](C(C)C)NC(=O)[C@H](CCCN=C(N)N)NC(=O)[C@H](CC(=O)O)N |
| Formula: | C50H71N13O12 |
| M.Wt: | 1046.179 |
| Purity: | >98% |
| Sotrage: | 2 years -20°C Powder, 2 weeks 4°C in DMSO, 6 months -80°C in DMSO |
| Description: | Angiotensin II human is a peptide hormone that causes vasoconstriction and a subsequent increase in blood pressure. In nature, angiotensin II (Ang II) is formed by the action of angiotensin converting enzyme (ACE), which cleaves the C-terminal -His-Leu from the decapeptide, angiotensin I. Angiotensin II is a vasoconstrictor, while its precursor is inactive. It is important in regulating cardiovascular hemodynamics and cardiovascular structure. Most of the known effects of Ang II in adult tissues are attributable to the angiotensin II type I (AT1) receptor. The AT1 and AT2 receptors have differential pharmacological and biochemical properties, and appear to exert opposite effects in terms of cardiovascular hemodynamics and cell growth. In addition, the renin-angiotensin and nitric oxidegenerating systems appear to interact in the regulation of cardiovascular function. Ang II stimulates angiogenesis and increases microvessel density. |
MSDS
COA
| LOT NO. | DOWNLOAD |
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| 2018-0101 |
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